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The intended therapeutic effect of gonadotropin-releasing hormone GnRH agonists is hypogonadism, a major cause of acquired osteoporosis in men. Consistent with this observation, GnRH agonists increase bone turnover and decrease bone mineral density, a surrogate for fracture risk. Large claims-based analyses and other retrospective studies provide compelling evidence that GnRH agonists increase risk of clinical fractures. Estrogens play a central role in homeostasis of the normal male skeleton, and estrogen deficiency rather than testosterone deficiency seems to be primarily responsible for the adverse skeletal effects of GnRH agonists. In randomized controlled trials, bisphosphonates pamidronate and zoledronic acid and selective estrogen receptor modulators raloxifene and toremifene increased bone mineral density in GnRH agonist—treated men.
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He was 67 when he was diagnosed in November, His initial PSA was 6. His choice of treatment was Surgery Robotic Laparoscopic Prostatectomy. I started yearly PSA testing in My PSA stayed about the same for the first few years, then it started to climb. Neither worked.
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Events include emotional symptoms such as crying, irritability, restlessness impatience, anger, and acting aggressive. You are encouraged to report negative side effects of prescription drugs to the FDA. You should notify your doctor if you experience fluid retention, seizure, https://www.billingsclinic.com asthma or worsening of asthmatic symptoms, or heart or kidney problems. Since some conditions may increase the possibility of bone thinning, you should tell your doctor if you smoke, use alcohol in excess, have a family history of osteoporosis thinning of the bones with fractures, or are taking other medications that can cause thinning of the bones. Even though you may not have periods, unprotected intercourse could result in pregnancy.
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Gonadotropin-releasing hormone agonists decrease bone mineral density, lean mass, and muscle size and increase fat mass in men with prostate cancer.
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